Download Bioceramics and Alternative Bearings in Joint Arthroplasty: by Jun-Dong Chang, Karl Billau PDF

By Jun-Dong Chang, Karl Billau

This complaints ebook of the Biolox Symposium in Seoul consists of 10 periods and plenary lectures of the most up-tp-date wisdom to be had within the use of Bioceramics and replacement bearings. greater than 50 audio system with world-famous reputations from 12 international locations hide fifty two issues on contemporary advancements in Bioceramic and substitute bearings in arthroplasty.

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Read Online or Download Bioceramics and Alternative Bearings in Joint Arthroplasty: 12th BIOLOX® Symposium Seoul, Republic of KoreaSeptember 7 - 8, 2007Proceedings (Ceramics in Orthopaedics) PDF

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Extra info for Bioceramics and Alternative Bearings in Joint Arthroplasty: 12th BIOLOX® Symposium Seoul, Republic of KoreaSeptember 7 - 8, 2007Proceedings (Ceramics in Orthopaedics)

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2004] Localization of RANKL in osteolytic tissue around a loosened joint prosthesis. J Bone Miner Metab, 22, 346-51. 32. Ikeda F, Nishimura R, Matsubara T, et al. [2004] Critical roles of c-Jun signaling in regulation of NFAT family and RANKL-regulated osteoclast differentiation. J Clin Invest, 114, 475-84. 33. Ingham E, Fisher J [2005] The role of macrophages in osteolysis of total joint replacement. Biomaterials, 26, 1271-86. 34. Iotsova V, Caamano J, Loy J, Yang Y, Lewin A, Bravo R [1997] Osteopetrosis in mice lacking NF-kappaB1 and NF-kappaB2.

Dev Cell, 8, 751-64. 17. Goldring SR, Goldring MB [2007] Eating bone or adding it: the Wnt pathway decides. Nat Med, 13, 133-4. 18. Goodman SB, Ma T, Chiu R, Ramachandran R, Smith RL [2006a] Effects of orthopaedic wear particles on osteoprogenitor cells. Biomaterials, 27, 6096-101. 19. Goodman SB, Ma T, Spanogle J, et al. [2006b] Effects of a p38 MAP kinase inhibitor on bone ingrowth and tissue differentiation in rabbit chambers. J Biomed Mater Res A, 81A, 310-316. 20. Granchi D, Amato I, Battistelli L, et al.

AP-1 become activated after Ti treatment of macrophages and fibroblast [20,39]. By an as yet unknown mechanism, RANKL induces the expression of the proto-oncogene c-Fos [32,63]. The expression of the c-fms gene, which encodes the receptor for M-CSF, is directly linked to the activation of the receptor for the granulocyte/macrophage colony-stimulating factor (GM-CSF). The binding of M-CSF to the ‘c-fms’ receptor triggers a signal transduction pathway which promotes the growth and survival of the committed OCPs, and up-regulates the RANK expression, thus making them available for the RANKL stimulus [20].

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